Hi,
This is a summary of the situation the last 6 months.
This first section is being written on April 14, 2004. For just the
last three months skip to July 7, 2004.
A PSA April 7 was up to 10.8 from 6.2. ?the main regimen has not
changed.10.8
A DRE seemed to indicate a regression in the PCa rather than an
increase.
It is quite possible by this time that the aggressiveness has increased.
It has
been 13 years and 3 months since the original diagnosis. ?I was told at
the
time
that the cancer was not the most aggressive but not slow
growing. ?Gleason
scores were not available from that lab at that time.
In the book, "The Omega Plan" by Drl Artemis P Simopoulos, it is pointed
out
that one of the effects of Omega 3 is the inhibition of metastasis. ?If
PCa
can
be kept from metastasizing there is no great danger. ?The original
metastasis
to the lymph glands has not been in eavidence since late 1991.
The Oncologist noted that there are no symptoms and he did not seem to
feel
there was any cause for alarm.
There is going to be one addition as a result of reading the attachment
below.
Cliff
*
The University of Virginia research determined that the prostate
cancer cells will only eat a chemical called 5-HETE."
the cancer center determined that 5-HETE was created from the
body's 'combustion' of an Omega-6 fatty acid called arachidonic acid.
There is an enzyme called 5-lipoxygenase (or 5-LO for short), and
when 5-LO burns or oxidizes the fatty acid, out plops 5-HETE."
the researchers concluded that prostate cancer cells eat ONLY this
food, and that if the 5-LO enzyme is inhibited, and the food is not
around, then the prostate cancer cells experience massive and
repaid 'apoptosis,' or cellular suicide, within I to 2 hours."
ginger is a powerful inhibitor of the 5-LO enzyme."
ginger is the world's most comprehensive herbal inhibitor of 5-LO,
with at least 22 proven phytonutrient inhibitors of that enzyme. What
is more, Oriental cultures that regularly consume ginger as a spice,
or which take it therapeutically, experience a fraction of the cancer
mortalities compared to the United States. In other words, men don't
die from prostate cancer cells we die from the growth and
proliferation of the cancer. Once we know that ginger inhibits the
process that leads to the creation of the ONLY food the cancer cells
will eat, it gives us confidence that an herbal approach to 5-LO
inhibition is an indispensable part of prostate health
the cancer cells only eat 5-HETE, ?and without it they begin to
massively and rapidly die within one to two hours."
Read More
P.M.
****
July 2004
I am now further updating on July 20. 2004.
A PSA taken on July 7, 2004 showed a spike from 10.8 on April 7, 2004
to 23.2.
PSA April 7, 2004 10.8
PSA July 7, 2004 23.2
This was a bit of a disappointment to say the least. Things like this
have happened before. After the initial surprise I try to analyze the
situation and try to figure what went wrong.
After really looking at it I don't believe it is hard to figure. For
the last four or five months I have been a little careless concerning
proper diet. I know that, while I haven't gone "hog wild" on sweets I
haven't exactly abstained. I haven't followed the Budwig Protocol
nearly as closely as I could have.
From this day on I am incraeasing the FO/CC to 5 tablespoons a day. I
am also going to a diet that will exclude most of the things that need
to be excluded. I need to get more rest. If one is going to control
PCa with a nutritional approach one cannot go just half way.
I have been using ellagic acid and powdered ginger. Apparently they
have not been of major help as yet.
I understand that one complete nutrition package is brown rice and beans
cooked without sugar. Just boil them and season. I did it tonight and
I really liked it. A little chicken gravy on the rice fixed it up.
I will follow this for three months to the next PSA. We need to know
whether or not this approach can be used to control an at least semi-
aggressive PCa. For many with aggressive, refactory PCas there is
little else that promises any real help. I will continue to be a
clinical trial of 1.
